He stayed in the hospital less than 24 h, i.e., much less than other pneumorachis cases this website reported in the literature.
He was actually discharged immediately after the CT-scan performed at 24 h had shown stability, while previously reported cases admitted for pneumomediastinum or pneumorachis were monitored for much longer. No oxygen was delivered, which is atypical since many patients with pneumothorax or pneumomediastinum are treated with oxygen to achieve nitrogen washout [5]. This observation thus suggests that patients admitted for pneumorachis after cocaine sniff-induced intra-bronchial hyper-pressure can be monitored for a short period of time and do not require oxygen therapy. Additional observations are required to confirm these findings. Pneumomediastinum has been recognized several decades
ago as a non life-threatening complication of cocaine sniffing. Association with a pneumorachis in the same patient had not been reported so far. Treatment consists in a simple monitoring during a short period of time. “
“It has been reported that Human metapneumovirus (hMPV) was associated with various upper and lower respiratory tract Selisistat concentration syndromes, including common colds, bronchitis, pneumonia, and asthma exacerbation, with more severe diseases reported for young children, elderly subjects, and immunocompromised patients [1], [2] and [3]. In adults, large outbreaks of hMPV infection in long-term care facilities (LTCF) have been reported, Carnitine dehydrogenase the magnitude and severity of which were similar to those of outbreaks typically associated with influenza or RS virus
infection [4] and [5]. Boivin et al. reported that 9 (9.4%) of 96 patients in the LTCF died due to respiratory infection, including 3 patients who had confirmed hMPV infection [4]. In this report, we found a severe respiratory failure case due to co-infection hMPV and Streptococcus pneumoniae in adult patient, and this case was diagnosed by not only routine microbiological methods and but also genetic analysis, including next-generation sequencer. A 64-year-old male patient who had been followed up for mild dilated cardiomyopathy became dyspneic and was admitted to our hospital in March 2013. He had been febrile (37.5 °C) for 1 week and had been coughing and short of breath for 2 days although he was a nonsmoker. His physiological parameters upon admission were as follows: blood pressure 94/52 mmHg, respiratory rate 24 breaths/min and PaO2 62 mmHg, despite the immediate administration of supplemental oxygen (10 L/min). Laboratory studies revealed no leukocytosis (white blood cells, 3500/mL), but its differentiation was as follows: Neu 87.5%, Lym 7.6%, Mono 3.8%, Eo: 0.6%, and Baso 0.4%, respectively, and inflammation was indicated by C-reactive protein 26.79 mg/dL (<0.8 mg/dL). In addition, aspartate transaminase 291 U/L and alanine transaminase 381 U/L; mild renal dysfunction was indicated by creatinine 1.18 mg/dL and blood urea nitrogen59 mg/dL.